Regulation of gene expression
A. Introduction
- Need
for regulation
- Types
of regulation
- negative regulation involves a repressor protein
- positive regulation involves an activator
protein
B. The lac operon as a paradigm of negative regulation
- diauxie
was first evidence of de-repression
- two
genes -lacZ and lacY, and their products, beta-galactosidase and permease,
were shown to be induced by lactose
- constitutive
mutants, that showed unregulated levels of these enzymes, were isolated
- constitutive in this context means that the
enzyme levels were always high, whether or not lactose was present
- remember that this is a mutant phenotype (and
genotype). The original notation was to call the wild-type, inducible
genotype i+ and the constitutive mutant i-.
- today, we use the term constitutive to mean
"unregulated" (and it doesn't imply a mutant genotype).
|
Strain
|
Phenotype
|
|
i+Z+Y+
|
lacZ and lacY are inducible
|
|
i-Z+Y+
|
lacZ and Z are constitutive
|
|
i+Z+Y+/ i-Z-Y-
|
inducible (so, i- is recessive, see?)
|
C. Gratuitous inducers were synthesized by Lederberg
- these
are chemical analogs of lactose that can induce the lac operon, but are
not metabolized by the cell.
- examples,
IPTG (isopropyl thiogalactoside) and TMG (thiomethyl galactoside).
- They
were useful to study the induction process without complications caused by
the metabolism of the inducer.
D. PaJaMo (Pardee, Jacob, Monod; 1959)
- performed
a series of matings (conjugations) with strains of E. coli that contained
different alleles of lacI and lacZ.
- one
of the most informative matings was: F'i+z+/i-z-
- with this type of mating, you can actually do a
time course
- two time courses, one with inducer (TMG) and one
uninduced, were done.
- the phenotype of the merodiploid gradually
switched from constitutive to induced over the course of a 2h period.
- this proved that "…the i gene in its
active form controls the synthesis of a product which, when present in
the cytoplasm, prevents the synthesis of beta-galactosidase and
galactoside-permease, unless inducer is added externally…".
E. The operator (Jacob and Monod, 1960)
|
Genotype
|
phenotype
|
|
i+o+z+
|
inducible
|
|
i-o+z-(y-)/i+ocz+(y+)
|
constitutive (for LacZ and LacY); therefore, oc
is dominant and pleiotropic
|
|
i+o+z-/i+ocz+
|
constitutive
|
|
i+o+z+/i+ocz-
|
inducible ; therefore, cis acting
|
|
i+o+z-y+/i+ocz+y-
|
constitutive for LacZ, inducible for LacY
|
1.
characterized
another type of constitutive mutation, called oc.
2.
"The constitutive
mutation oc is thus pleiotropic and dominant, and its effect is only
manifested in the cis position."
3.
P.S. The operator was
actually discovered before the promoter.
F. the Operon Concept
Positive regulation of the lac operon
A. back to diauxie
- What
dictates the pattern in which glucose is used first, lactose is used only
after glucose is gone?
- What
is the state of the lac operon in the various phases of the diauxic growth
curve?
B. catabolite repression is the term for the phenomenon that underlies the
preference of bacteria for glucose (or any particular growth substrate).
C. cAMP and adenylate cyclase
D. CRP (the cAMP receptor protein)
- CRP
is inactive by itself
- CRP-cAMP
is able to bind near the lac promoter
- converts
it from a weak to a strong promoter
E. the lac promoter is not strong by itself
- 5'
TATGTT 3' is not a good match to consensus
- however,
when CRP-cAMP binds to it, it becomes stronger.
- the
lacUV5 promoter contains a double mutation that converts it from 5'TATGTT
3' to 5' TATAAT 3'
- is this promoter still subject to negative
regulation?
- is it still subject to positive regulation?
- email your
answer for zillions of bonus points
Summary of regulation of lac operon
A. Return to diauxie
- during
first exponential phase, lac repressor is inactive, but so is CRP, so
transcription of lac operon is minimal
- during
middle plateau, cAMP builds up, and activates CRP. The active
complex binds and stimulates transcription of lacZYA. Transcription
is maximal, because both activation (CRP-cAMP) and derepression (lacI
de-activation) are at work.
B. catabolite repression operates on several metabolic operons (maltose,
arabinose, galactose, etc.), not just lac. It's a general effect of
glucose, which is the mother of all growth substrates for E. coli.
C. try this one: what is the state of the lac operon in cells growing on
maltose only? On maltose plus lactose?
Attenuation at the trp operon
a. Different
modes of regulation: catabolic vs. anabolic
1.
"Classical"
negative regulation
2.
like lac, involves a
repressor
3.
unlike lac, repressor is
inactive as default state, becomes active in presence of the co-repressor
tryptophan.
4.
genetic example of
feedback inhibition
b. attenuation
1.
this is a mechanism to
achieve fine control over trp operon expression
1.
is a example of the
tight coupling of transcription and translation in bacteria.
2.
involves a stretch of
DNA called the attenuator region
3.
region contains
transcriptional and translational signals
4.
translation signal is a
short open reading frame that features two back-to-back trp codons (UGG UGG).
2.
think what will happen
to translation when tryptophan is scarce in the cytoplasm.
1.
transcriptional signal
consists of two possible stem loop structures: one is a terminator, and the
other is an "antiterminator".
2.
if tryptophan is abundant,
translation occurs without pause, and this favors the formation of the
terminator. Transcription of downstream structural genes (trpEDCBA)
doesn't happen.
3.
if tryptophan is scarce,
translation of the leader pauses at UGG UGG, and this favors the formation of
the antiterminator. Transcription of trpEDCBA occurs..